Cardiac Pharmacology by R. Douglas Wilkerson (Eds.)

By R. Douglas Wilkerson (Eds.)

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This points out the need for a beta blocking agent that counteracts sympathetic stimuli influencing cardiac electrical but not cardiac mechanical activity. That such an approach might be possible is indicated from the report of Bonelli (1978). This investigator describes a selective blocking effect of mepindolol sulfate on cardiac ß 1 receptors mediating changes in heart rate as opposed to receptors mediating changes in cardiac contractile force. Drugs that either activate or inhibit vagal influences on the heart also influence cardiac rhythm disorders.

Digitalis Neural effects of digitalis drugs are important in treating cardiac arrhythmias (Gillis and Quest, 1978). In terms of treating atrial arrhythmias, their effect to slow ventricular rate in patients with either atrial flutter or fibrillation results 2. Drugs Affecting Cardiovascular Function 35 from neurally mediated changes in A-V conduction. Some of the slowing in ventricular rate that occurs in atrial flutter is due to the development of atrial fibrillation and enhancement of concealed conduction.

1976). The CNS mechanism(s) whereby clonidine exerts its antihypertensive effect appears to be due to activation of α-adrenergic receptors. Evidence for this has been summarized in the two recent reviews on this drug (Schmitt, 1977; Kobinger, 1978). The question of whether this involves activation of pre- or postsynaptic α-adrenergic receptors is not fully resolved. However, it has been reported that clonidine produces the same reduction in sympathetic nerve activity in animals depleted of CNS catecholamines by coadministration of reserpine and a -methyl-/?

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