By W. Burggren, B. Keller
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Extra resources for Devel. of Cardiovascular Systs - Molecules to Organisms
Atherosclerotic cerebrovascular disease is a common cause of strokes and shows a predilection for sites such as the bifurcation of the common carotid artery into the internal and external carotid arteries and the aortic arch and the major intracranial arteries such as the basilar artery and the middle cerebral arteries. Occlusive atherosclerotic vascular disease of these large extracranial arteries is responsible for as many as 20–30% of ischemic strokes and intracranial steno-occlusive disease causes around 5–10% of ischemic strokes.
The serine protease tissue-type plasminogen activator [TPA], the utility of which is limited by the short therapeutic window. As new targets are identified, new opportunities emerge that build on an appreciation of acute cellular events acting in a broader context of ongoing destructive, protective, and reparative processes. At the onset of the 21st century, it is the third-leading cause of death in most developed countries and the primary cardiovascular cause of death in Japan and China. The health burden of the disease is staggering as loss of a productive life inflicts a heavy toll on patients, families, and society.
Potential advantages to this approach may include greater control over cell fate, the ability to deliver any desired number of cells and reduced risks associated with mitogen 24 Manzoor A. Mir infusion. The major goal is reconstitution of the complex and widespread neuronal–glial– endothelial interrelationship may require access to a broader array of cell lineages, since stroke affects multiple cell types including neurons, glia and endothelial cells. Thus, ideally, cells should need to maintain initially an immature state and differentiate into several specific cell types after engraftment.